Share Success: Letters From Readers
I: From Linda H.
Submit Form Tuesday, March 10, 2015 4:28 PM
I have been taking Ezorb for 5 weeks and have seen many positive changes in the way my neck feels and moves. My neck feels more flexible and not as sore.
I have arthritis in my neck and possible bone spurs and maybe a herniated disc. Have not been able to get an MRI so don't know this definitely. I do have one problem area that is located near the neck joint. I suspect I may have bone on bone between two neck vertebrae because of the burning sensation and soreness in that area after use.
II: From Lon E.
Submit Form Saturday, January 31, 2015 at 10:21:01
Hi, my name is Lon E. and I had Bone spurs in both elbows.
My Doctor (Mayo Clinical) wanted to do surgery, but my wife said for me to wait. She was praying for a cure. She was on the Internet and researching different products, and came across a testimonial regarding heel spurs being cured.
She asked if I would try this product before surgery. It was Ezorb. Boy am I glad I did. You need to know I couldn't shave my face, write, very difficult to brush my teeth, couldn't straighten my arms or lift them very high. Sleep was very difficult.
After about two weeks things started to improve. 3 weeks later my wife and I were going out to eat and I started clapping my hands. My wife ask what I was doing that for and I said, because I can.
Thank you for your product and if anyone with bone spurs reads this please try it.
Las Vegas, NV
III: From Anonymous
Submit Form Wednesday, December 31, 2014 at 11:40:58
Been using Ezorb Calcium for 8 years after diagnosis of Osteoporosis. Against medical advice, I might add; this calcium has literally saved my life by preventing serious injury from a fall in a public place due to carelessness by store personnel, and on another
occasion when my young (and very large) dog when she bolted in fright. My bones are strong , not brittle.
And Marvlix has been Marvelous for me. Take it regularly and have not developed bronchitis since I began almost 8 years ago, as well. I cannot say enough about
your two products! Except THANK YOU!!!
the Desk of EZorb Newsletter Editor:
newsletter is now read by over 70,000 subscribers
worldwide. Success stories you have contributed over
the years have had a great impact on many people's
quality of life. Your continuous support will be
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suffering and would continuously suffer, without your
help! Please email your
story to sharesuccess @ ezorbonline.com
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Research News: Brain Iron Level May Be Related to Alzheimer's Risk
Ferritin in the cerebrospinal fluid (CSF) of patients with Alzheimer's disease (AD) is associated with the APOE e4 risk allele and predicts cognitive outcomes, a study shows.
"Collectively these data support consideration of therapeutic strategies that lower brain iron, which have reported beneficial outcomes in Phase II trials of Alzheimer's and Parkinson's diseases", write the study authors in Nature Communications.
"Lowering CSF ferritin, as might be expected from a drug like deferiprone, could conceivably delay MCI [mild cognitive impairment] conversion to AD by as much as 3 years."
Ashley Bush (The University of Melbourne, Victoria, Australia) and colleagues measured baseline CSF levels of ferritin, as an indicator of brain iron levels, in 302 participants of the US-based Alzheimer's Disease Neuroimaging Initiative.
Cortical iron levels are reportedly elevated in AD, which, it is thought, may contribute to cognitive decline. In the current study, the team found that ferritin levels were related to participants' baseline cognitive scores on the Alzheimer's Disease Assessment Scale, with levels in the top tertile (>7.2 ng/mL) associated with about a 3-point lower score compared with levels in the bottom tertile (<5.4 ng/mL).
Ferritin also predicted changes over time, but its effect on cognitive function was the same at all time points, identifying it as a trait variable, in contrast to the effects of disease markers such as APOE e4, which increased over time.
Because of this constant effect, people with MCI who had high ferritin levels met the criteria for AD earlier than those with lower levels, with each standard deviation increase resulting in a 9.5-month shift to an earlier age of conversion. This compared with 18.2- and 8.6-month shifts for a standard deviation increase in ApoE and the ratio of tau to B-amyloid (AB)1–42, respectively.
Adding ferritin to a model containing these two markers plus age, gender, education and body mass index increased the model's predictive accuracy for conversion to AD "markedly", from 64.83% to 69.37%.
The potential underlying reason for the effect of ferritin on cognitive outcomes came when the team detected "an unexpected interaction of ApoE with ferritin."
They found that CSF ApoE level accounted for 24.3% of the variability in CSF ferritin when tau and AB1–42 were included in the model and 34.1% when they were removed.
Moreover, CSF ferritin levels were 22% higher in carriers of the APOE e4 risk allele than in noncarriers, while CSF ApoE levels were 16% lower.
"That ferritin levels are increased by the APOE-e4 allele argues that ApoE influences ferritin levels, rather than the reverse", observe the researchers.
They speculate "that the e4 genotype raises the baseline iron load of the brain, thus lowering the threshold for iron-mediated neuronal loss in disease."
Original research was published in Nat Commun 2015; Advance online publication.
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